Sucralfate

證據等級: L5 預測適應症: 2

目錄

  1. Sucralfate
  2. Sucralfate: From Peptic Ulcer Disease to Duodenogastric Reflux
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Safety Considerations
    7. Conclusion and Next Steps
    8. Disclaimer

## 藥師評估報告

Sucralfate: From Peptic Ulcer Disease to Duodenogastric Reflux

One-Sentence Summary

Sucralfate (DrugBank DB00364) is a mucosal-protective agent historically used in the management of peptic (duodenal/gastric) ulcer disease, based on the drug's own literature record rather than a New Zealand regulatory filing, since the drug currently holds no marketing authorization in New Zealand. The TxGNN model predicts it may be effective for Duodenogastric Reflux, with 0 registered clinical trials and 13 relevant publications currently supporting this direction. A second candidate indication, duodenal obstruction, scored similarly (99.30%) but has even thinner supporting literature and is not covered further below.


Quick Overview

Item Content
Original Indication Not documented in New Zealand regulatory records (drug not marketed); literature in this evidence pack references historical use in duodenal/gastric ulcer and erosive gastroduodenitis
Predicted New Indication Duodenogastric reflux
TxGNN Prediction Score 99.37% (rank 5283)
Evidence Level L3
New Zealand Market Status ✗ Not Marketed
Number of Authorizations 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Detailed mechanism-of-action data is not available in this evidence pack (data gap). Based on the retrieved literature itself, sucralfate is described as having "cytoprotective features in addition to its known antipepsin and antacid effects" (PMID 3838414), and case/randomized studies describe its use for mucosal protection against gastric erosion and ulceration.

Duodenogastric reflux involves retrograde flow of alkaline duodenal (bile-containing) content into the stomach, producing mucosal injury (alkaline/bile reflux gastritis) that is mechanistically related to the acid-peptic mucosal injury sucralfate is traditionally used to treat. Multiple retrieved publications directly studied sucralfate for this indication — including randomized trials against placebo (PMID 3475771) and against rabeprazole (PMID 12923369) — which is consistent with the TxGNN prediction and suggests the model captured a genuine, previously-studied mechanistic link rather than a purely novel association.

Because taiwan_regulatory contains no New Zealand licenses for sucralfate, this rationale relies on literature evidence within the pack rather than an official approved-indication text, and should be treated as lower-confidence until a formal MOA and regulatory record are obtained.


Clinical Trial Evidence

Currently no related clinical trials registered


Literature Evidence

PMID Year Type Journal Key Findings
3839973 1985 RCT The American Journal of Medicine Randomized, double-blind study of sucralfate 6g/day vs. placebo in 23 patients with alkaline reflux gastritis symptoms after Billroth I/II or vagotomy/pyloroplasty
12923369 2003 RCT European Journal of Gastroenterology & Hepatology Randomized trial of sucralfate vs. rabeprazole vs. no treatment for post-cholecystectomy alkaline reactive gastritis
3475771 1987 RCT Scandinavian Journal of Gastroenterology. Supplement Prospective randomized trial of sucralfate vs. placebo in patients with symptomatic/macroscopic gastritis, including duodenogastric reflux comparison
1391144 1992 Comparative study Minerva Gastroenterologica e Dietologica Compared cisapride (prokinetic) vs. sucralfate (cytoprotective) for dyspeptic symptoms in 18 patients with duodenogastric reflux gastritis
17285081 2006 Review Journal de Chirurgie Review of duodenogastric and gastroesophageal bile reflux pathophysiology, diagnosis (24h bile monitoring), and therapeutic management
14723838 2004 Review Current Treatment Options in Gastroenterology Review of duodenogastric reflux-induced (alkaline) esophagitis; notes PPIs as best medical treatment, difficulty of DGER management
6372664 1984 Review Annual Review of Medicine Review of alkaline reflux (bile) gastritis and esophagitis, pathophysiology and diagnostic features
3552846 1987 Review Gastroenterologie Clinique et Biologique Pharmacologic basis of medical treatment for duodenogastric reflux (abstract not available)
3838414 1985 Review The American Journal of Gastroenterology ACG committee review of sucralfate's non-ulcer uses, including cytoprotective, antipepsin, and antacid effects
3616071 1987 Case series Revista Española de las Enfermedades del Aparato Digestivo 50 cases of postsurgical biliary reflux gastritis treated with sucralfate (abstract not available)

Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Hold

Rationale:

  • Sucralfate is currently not marketed in New Zealand (0 licenses) and package-insert-level safety/warning data is marked as a Blocking data gap, so the candidate cannot yet pass an initial safety screen; supporting evidence for duodenogastric reflux is limited to small, unregistered historical trials and reviews rather than modern registered clinical trials.

To proceed, the following is needed:

  • TFDA/manufacturer package insert (warnings, contraindications, DDI) to clear the Blocking data gap (DG001)
  • Confirmed drug MOA from DrugBank or equivalent source (DG002)
  • Evaluation of whether a New Zealand marketing pathway exists for sucralfate before further repurposing investment
  • A contemporary, registered clinical trial (ClinicalTrials.gov/ICTRP) confirming efficacy in duodenogastric reflux, since none currently exist

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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