Risperidone

證據等級: L5 預測適應症: 6

目錄

  1. Risperidone
  2. Risperidone: From Psychotic Disorders to Major Affective Disorder
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. New Zealand Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## 藥師評估報告

Risperidone: From Psychotic Disorders to Major Affective Disorder

One-Sentence Summary

Risperidone is a widely known atypical antipsychotic (D2/5-HT2A antagonist) generally used for schizophrenia and bipolar disorder — no Taiwan/NZ regulatory indication text is on file for this drug in this evidence pack. Among 6 TxGNN-predicted indications, Major Affective Disorder (depression/bipolar spectrum) is by far the strongest candidate, supported by 37 clinical trials (including multiple completed Phase 3 RCTs directly testing risperidone) and 20 publications, several of which are systematic reviews/meta-analyses of risperidone augmentation in depression and bipolar disorder. The remaining 5 predicted indications range from weakly indirect (Asperger susceptibility, Phelan-McDermid syndrome, trichotillomania) to mechanistically implausible (gaze palsy with scoliosis, amelocerebrohypohidrotic syndrome) and are not pursued as the headline finding.


Quick Overview

Item Content
Original Indication Not available on file (see note below)
Predicted New Indication Major Affective Disorder
TxGNN Prediction Score 99.11%
Evidence Level L1
New Zealand Market Status ✗ Not Marketed
Number of Authorizations 0
Recommended Decision Hold

Note on Original Indication: No Taiwan/NZ license or indication text exists in the regulatory data provided (market_status: 未上市, 0 licenses, original_indications: []). The original-indication context above (schizophrenia/bipolar disorder) reflects general public drug knowledge, not data extracted from this evidence pack, and should be confirmed against a formal source before use in any regulatory submission.


Why is This Prediction Reasonable?

Detailed mechanism-of-action data is not available in this evidence pack (original_moa: [Data Gap]). Based on generally known pharmacology, risperidone is a potent dopamine D2 and serotonin 5-HT2A receptor antagonist ("atypical antipsychotic"), a receptor profile long associated with mood-stabilizing and antidepressant-augmenting effects in addition to its antipsychotic action.

Major affective disorder (encompassing bipolar disorder and treatment-resistant major depressive disorder, MDD) sits directly adjacent to risperidone's core pharmacological class. Risperidone is already an established treatment for bipolar mania in several jurisdictions, and its use as an adjunct to SSRIs/SNRIs in treatment-resistant depression is supported by multiple completed Phase 3 randomized controlled trials in this evidence pack (e.g., NCT00095134, NCT00044681) plus several systematic reviews and meta-analyses of second-generation antipsychotic augmentation in MDD. This is a mechanistically direct and clinically well-precedented link, not merely a computational artifact.

For context, the top-ranked TxGNN prediction overall ("gaze palsy, familial horizontal, with progressive scoliosis," score 99.76%) is explicitly flagged in this evidence pack's own rationale as having "no known mechanistic or clinical link" to risperidone's receptor pharmacology — a rare ROBO3-mutation brainstem axon-guidance disorder — and returned zero trials and zero literature. "Amelocerebrohypohidrotic syndrome" (rank 3) is similarly a rare ectodermal dysplasia with no plausible pharmacological connection and no evidence. "Asperger syndrome, susceptibility to" (rank 2) and "Phelan-McDermid syndrome" (rank 4) have indirect support only — risperidone is used off-label for irritability/behavioral symptoms in autism-spectrum populations broadly, but no disease-specific trials or strong literature exist for either entity in this pack. "Trichotillomania" (rank 5) has 10 case-report/case-series-level publications (L3) showing SSRI-augmentation benefit but no RCTs. None of these four match the strength of evidence behind Major Affective Disorder, so this report focuses on that indication.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT00095134 Phase 3 Completed 630 Adjunctive risperidone vs. placebo in MDD with sub-optimal response to standard antidepressant therapy
NCT00044681 Phase 3 Completed 258 Risperidone augmentation of SSRI monotherapy in young and older adults with unipolar treatment-resistant depression, including long-term maintenance effect
NCT00391222 Phase 3 Completed 585 Risperidone long-acting injectable monotherapy vs. placebo for prevention of mood episodes in bipolar I disorder
NCT00277654 Phase 3 Completed 111 Risperidone monotherapy vs. placebo in ambulatory bipolar disorder with comorbid panic disorder/generalized anxiety disorder
NCT00167479 Phase 4 Completed 60 Risperidone monotherapy efficacy/tolerability in bipolar disorder with moderately severe anxiety
NCT00107939 Phase 3 Completed 453 Adjunctive therapy trial in bipolar mania where risperidone was one of five background atypical antipsychotics studied
NCT00057681 Phase 3 Completed 379 TEAM study: lithium vs. valproate vs. risperidone in children/adolescents with bipolar disorder or mania symptoms
NCT00571688 Phase 4 Completed 50 Risperidone Consta (LAI) vs. treatment-as-usual for reducing relapse/rehospitalization in bipolar disorder
NCT01282632 Phase 1/2 Completed 42 Pilot trial of risperidone vs. olanzapine as add-on to a failed SSRI in treatment-resistant depression
NCT00203723 Phase 4 Terminated 45 Combined ECT + risperidone vs. ECT alone for treatment-resistant depression

Literature Evidence

PMID Year Type Journal Key Findings
17975181 2007 RCT Annals of Internal Medicine Randomized trial of risperidone for treatment-refractory major depressive disorder
25295435 2014 Population-based study Journal of Clinical Psychiatry Nationwide effectiveness study of aripiprazole/olanzapine/quetiapine/risperidone augmentation for MDD
21154393 2010 Cochrane systematic review Cochrane Database of Systematic Reviews Second-generation antipsychotics (incl. risperidone) for major depressive disorder and dysthymia
34986373 2022 Systematic review + network meta-analysis Journal of Affective Disorders Compares efficacy/discontinuation of augmentation agents, including risperidone, in treatment-resistant depression
35861202 2023 Systematic review + meta-analysis Journal of Psychopharmacology Augmentation/combination treatments for early-stage treatment-resistant depression
35510505 2023 Systematic review + meta-analysis Psychological Medicine Efficacy and safety/tolerability of antipsychotics (monotherapy and adjunctive) in adult MDD
24919175 2014 Meta-analysis Brazilian Journal of Medical and Biological Research Efficacy/tolerability of antidepressant augmentation with atypical antipsychotics (17 trials, 3807 patients) in MDD
23554581 2013 Meta-analysis PLoS Medicine Adjunctive atypical antipsychotic treatment for MDD: depression, quality-of-life, and safety outcomes
21189367 2011 Review The Annals of Pharmacotherapy Efficacy and safety review of risperidone augmentation specifically for MDD
20486830 2010 Review Expert Opinion on Pharmacotherapy Risperidone long-acting injection as monotherapy and adjunctive therapy in maintenance treatment of bipolar I disorder

New Zealand Market Information

Risperidone is currently not marketed in New Zealand under this evidence pack's regulatory data source, with 0 authorizations on file. No product license records are available to summarize.


Safety Considerations

Please refer to the package insert for safety information. (Key warnings, contraindications, and drug-interaction data are all marked as data gaps in this evidence pack; the TFDA package insert warnings/contraindications item is flagged as a Blocking data gap — DG001 — that must be resolved before a formal safety assessment (S1) can proceed.)


Conclusion and Next Steps

Decision: Hold

Rationale: Efficacy evidence for risperidone in major affective disorder is strong (L1 — multiple completed Phase 3 RCTs plus several systematic reviews/meta-analyses), but the Blocking data gap on TFDA package insert warnings/contraindications (DG001) means a formal safety evaluation cannot yet be completed, and the drug has no current NZ market presence in this dataset.

To proceed, the following is needed:

  • TFDA/regulatory package insert warnings and contraindications (Blocking gap, DG001)
  • Confirmed original indication and MOA data from DrugBank or an authoritative label (DG002)
  • Formal DDI dataset (current query returned not_found)
  • Clarification of NZ market and licensing status, since no product is currently on file
  • Disease-specific evaluation of the four indirect/low-evidence predictions (Asperger susceptibility, Phelan-McDermid syndrome, trichotillomania) if pursued as secondary candidates

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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