Olaparib

證據等級: L5 預測適應症: 1

目錄

  1. Olaparib
  2. Olaparib: From Ovarian Cancer to Breast Cancer
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. New Zealand Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Olaparib: From Ovarian Cancer to Breast Cancer

One-Sentence Summary

Olaparib is a PARP1/2 inhibitor originally developed for maintenance treatment of platinum-sensitive relapsed, BRCA-mutated high-grade serous ovarian, fallopian tube, or peritoneal cancer. The TxGNN model predicts it may also be effective for Female Breast Carcinoma, a prediction already strongly corroborated by 80 clinical trials and 20 publications, including two landmark Phase III RCTs (OlympiAD, OlympiA) that have led to approved breast cancer indications for olaparib in other jurisdictions.


Quick Overview

Item Content
Original Indication Ovarian cancer — maintenance treatment of platinum-sensitive relapsed, BRCA-mutated high-grade serous epithelial ovarian/fallopian tube/peritoneal cancer
Predicted New Indication Female Breast Carcinoma
TxGNN Prediction Score 99.09%
Evidence Level L1
New Zealand Market Status ✗ Not Marketed
Number of Authorizations 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Olaparib is a PARP1/2 (poly ADP-ribose polymerase) inhibitor. It blocks base-excision repair of single-strand DNA breaks, which in tumour cells carrying BRCA1/2 germline mutations (i.e. homologous recombination deficiency, HRD) leads to synthetic lethality — the cancer cell, already unable to repair double-strand breaks via homologous recombination, cannot compensate for the loss of the base-excision pathway either, and dies. Normal cells retain functional homologous recombination repair and are largely spared.

Ovarian cancer and breast cancer share the same underlying genetic vulnerability: both are core BRCA1/2-associated malignancies, and BRCA-mutated tumours in either tissue rely on the same DNA-repair deficiency that olaparib exploits. This mechanistic overlap is why the TxGNN prediction is biologically plausible rather than a spurious network association — the drug does not need a breast-specific mechanism, only a BRCA/HRD-positive tumour, which occurs in both cancer types.

This is also not a purely theoretical extrapolation: olaparib has already progressed through Phase III confirmatory trials in breast cancer (OlympiAD for metastatic disease, OlympiA for high-risk early-stage adjuvant use) and holds approved breast cancer indications in other regulatory jurisdictions (FDA, EMA), which independently validates the repurposing rationale identified by the model.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT02282020 Phase 3 Completed 266 OlympiAD: Olaparib vs physician's choice chemotherapy in gBRCA-mutated, HER2-negative metastatic breast cancer
NCT00679783 Phase 2 Completed 99 Early AZD2281 (olaparib) study establishing response rate in BRCA-mutated/triple-negative breast cancer, foundational for later Phase III design
NCT04330040 Phase 4 Completed 202 Post-marketing real-world study in Indian patients with platinum-sensitive relapsed ovarian cancer and gBRCA1/2-mutated metastatic breast cancer
NCT02418624 Phase 1 Completed 25 Carboplatin-olaparib sequencing vs capecitabine as first-line therapy in BRCA1/2-mutated HER2-negative advanced breast cancer
NCT05498155 Phase 2 Active, not recruiting 50 Neoadjuvant olaparib monotherapy vs olaparib + durvalumab in BRCA-mutated, early-stage HER2-negative breast cancer
NCT03109080 Phase 1 Completed 24 Olaparib combined with radiation therapy in inflammatory/locoregionally advanced/metastatic or residual triple-negative breast cancer
NCT01623349 Phase 1 Completed 118 Oral PI3K inhibitor (BKM120/BYL719) plus olaparib in recurrent triple-negative breast cancer or high-grade serous ovarian cancer
NCT02624973 Phase 2 Active, not recruiting 200 PETREMAC: personalized treatment strategies in high-risk breast cancer using predictive biomarkers
NCT04041128 Early Phase 1 Completed 14 Pre-surgical window study of PARP inhibition on cellular/molecular changes in primary ovarian and breast cancer
NCT05209529 Phase 2 Withdrawn 0 Neoadjuvant olaparib + durvalumab for BRCA-associated triple-negative breast cancer (did not proceed)

Literature Evidence

PMID Year Type Journal Key Findings
36228963 2022 RCT Annals of Oncology OlympiA: overall survival results of adjuvant olaparib in gBRCA1/2-mutated, high-risk early breast cancer
34081848 2021 RCT New England Journal of Medicine OlympiA primary results: adjuvant olaparib reduces recurrence in BRCA1/2-mutated early breast cancer
28578601 2017 RCT New England Journal of Medicine OlympiAD primary results: olaparib shows antitumor activity in metastatic breast cancer with germline BRCA mutation
30689707 2019 RCT Annals of Oncology OlympiAD final overall survival and tolerability vs chemotherapy of physician's choice
36893711 2023 RCT European Journal of Cancer OlympiAD extended follow-up confirming survival and safety profile
33119476 2020 Phase 2 Trial Journal of Clinical Oncology TBCRC 048: olaparib activity in metastatic breast cancer with somatic BRCA or other homologous recombination gene mutations
34143979 2021 Phase 2 Trial Cancer Cell I-SPY2: durvalumab + olaparib + paclitaxel increases pathologic complete response in high-risk HER2-negative breast cancer
35163586 2022 Review International Journal of Molecular Sciences Mechanisms, biomarkers and emerging therapies (including PARP inhibitors) for chemotherapy-resistant triple-negative breast cancer
33710534 2021 Review Targeted Oncology Overview of PARP inhibitors, including olaparib, approved for BRCA-mutated HER2-negative breast cancer
39520738 2024 Phase 2 Trial Breast (Edinburgh) NOBROLA: olaparib monotherapy in HRD-positive, non-germline-BRCA-mutated advanced triple-negative breast cancer

New Zealand Market Information

Olaparib currently has no marketing authorization on record in New Zealand (0 licenses).


Cytotoxicity

Item Content
Cytotoxicity Classification Targeted therapy (PARP inhibitor) — not a conventional cytotoxic chemotherapy agent
Myelosuppression Risk Please refer to the package insert warnings and precautions (no drug-specific toxicity data in current evidence pack; anaemia, neutropenia and thrombocytopenia are class-recognized effects of PARP inhibitors)
Emetogenicity Classification Please refer to the package insert warnings and precautions
Monitoring Items CBC with differential, renal and hepatic function
Handling Protection Please refer to the package insert warnings and precautions

Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: The mechanistic rationale (BRCA/HRD-driven synthetic lethality) is strong, and evidence is already at L1 strength — two completed Phase III RCTs (OlympiAD, OlympiA) directly support olaparib's efficacy in BRCA-mutated breast cancer, and it holds approved breast cancer indications elsewhere. However, the drug is not currently marketed in New Zealand, and safety/regulatory data (package insert warnings, contraindications, MOA, DDI) are all outstanding, so guardrails are needed before advancing.

To proceed, the following is needed:

  • TFDA/Medsafe package insert (warnings, contraindications) — currently a blocking data gap for safety assessment
  • Confirmed mechanism of action data from DrugBank
  • BRCA1/2 or HRD biomarker testing pathway/protocol for patient selection
  • New Zealand market entry/registration assessment, since olaparib is not currently authorized

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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